Nonlinear ODE and Markov models capture non-commutative clinical effects of JAK2-then-TET2 versus TET2-then-JAK2 mutation order on gene expression, cell fractions, and age at diagnosis in myeloproliferative neoplasms.
The phenotypic equilibrium of cancer cells: From average-l evel stability to path-wise convergence
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Order-of-mutation effects on cancer progression: models for myeloproliferative neoplasm
Nonlinear ODE and Markov models capture non-commutative clinical effects of JAK2-then-TET2 versus TET2-then-JAK2 mutation order on gene expression, cell fractions, and age at diagnosis in myeloproliferative neoplasms.