A spatially-resolved, lipid-structured model of early atherosclerosis reproduces the observed internal lipid and macrophage peaks only when macrophage mobility drops steeply with lipid load while lifespan changes little.
Arteriosclerosis, thrombosis, and vascular biology 16(1), 4–11 (1996)
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A spatially resolved and lipid-structured model for macrophage populations in early human atherosclerotic lesions
A spatially-resolved, lipid-structured model of early atherosclerosis reproduces the observed internal lipid and macrophage peaks only when macrophage mobility drops steeply with lipid load while lifespan changes little.