MnBi4Te7 single crystals show a field-cooling-induced exchange bias with a training effect that also shifts the anomalous Hall loops, attributed by the authors to Mn-Bi antisite defects.
Intertwined magnetic phase driven exchange bias and its impact on the anomalous Hall effect in MnBi$_4$Te$_7$
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abstract
We report on the interplay between atomic scale inhomogeneity and competing magnetic phases and its effect on the anomalous Hall effect in the layered antiferromagnet MnBi$_4$Te$_7$, a natural superlattice hosting coexisting ferromagnetic and antiferromagnetic phases. Using a combination of scanning tunneling microscopy (STM), DC and AC magnetization, and magneto-transport measurements, we reveal that intrinsic Mn Bi antisite defects induce strong interlayer exchange coupling, giving rise to a robust exchange bias observed in both magnetic and Hall responses. The exchange bias undergoes a transition from asymmetric to symmetric behavior between 2 K and 6 K, indicating a temperature driven dynamical reconfiguration of interfacial spin structures. The training effect analysis revealed a stronger contribution of frozen spins at 2 K compared to 6 K, with relaxation amplitude shift from -264 Oe to 306 Oe. This sign reversal indicates a field-induced change in interfacial coupling. The temperature dependence of longitudinal resistivity and magnetization reveals complementary behavior, indicating the coexistence of two distinct spin states near the magnetic transition temperature. The phase fraction based resistivity model captures the distinct scattering mechanisms that govern electronic transport across different magnetic regimes. Our findings offer a direct link between microscopic disorder, interfacial magnetism and macroscopic topological phenomena in magnetic topological insulators.
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Intertwined magnetic phase driven exchange bias and its impact on the anomalous Hall effect in MnBi$_4$Te$_7$
MnBi4Te7 single crystals show a field-cooling-induced exchange bias with a training effect that also shifts the anomalous Hall loops, attributed by the authors to Mn-Bi antisite defects.