A pair of biophysical simulations links reduced ATP free energy to shorter-lived solitons in protein alpha-helices and to hyperexcitable CA1 pyramidal neurons, proposing a thermodynamic mechanism for mental fatigue.
Lower gene expression for KCNS3 potassium channel subunit in parvalbumin- containing neurons in the prefrontal cortex in schizophrenia
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Reduced Gibbs free energy supply hinders brain information processing during mental fatigue
A pair of biophysical simulations links reduced ATP free energy to shorter-lived solitons in protein alpha-helices and to hyperexcitable CA1 pyramidal neurons, proposing a thermodynamic mechanism for mental fatigue.