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arxiv: 2605.27580 · v2 · pith:GEIOFQKFnew · submitted 2026-05-26 · 💻 cs.AI · q-bio.NC

You Are in Control of Your State: Why Human Outcomes Are Controllable Through Causal State Intervention

Pith reviewed 2026-06-29 17:06 UTC · model grok-4.3

classification 💻 cs.AI q-bio.NC
keywords causal state interventiondynamic latent statewithin-person variabilitybehavioural controllabilitystate-aware systemsattentional bottleneckpredictive processingallostasis
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The pith

Human outcomes are controllable in a precise sense by intervening on the dynamic latent state at the moment a decision forms.

A machine-rendered reading of the paper's core claim, the machinery that carries it, and where it could break.

The paper claims that within-person variability in outcomes arises because the same input produces different results depending on a person's internal state at that instant. This state is a time-indexed weighting vector that shapes how biology, physiology, and neuropsychology turn an event into a decision and outcome. The relationship is presented as causal, so targeted interventions on the state and its weighting can steer results conditionally on the state trajectory. The argument draws on evidence from causal inference, predictive processing, allostasis, attention, chronobiology, and computational psychiatry, plus large-scale observational data. If correct, this reframes human-facing AI, digital health, education, and personal agency around state-aware intervention rather than fixed traits or observables alone.

Core claim

We argue that this variability belongs in the dynamic latent state of the person, and that human outcomes are controllable in a precise and operational sense through interventions that target the state and its weighting at the moment a decision is being formed. We define a state as the time-indexed weighting vector over the dimensions that govern how an individual's biology, physiology, and neuropsychology process the next event into a decision and an outcome. The relationship between state, decision, and outcome is causal rather than correlational. The weighting vector is dynamic at sub-daily timescales. The conscious channel through which outcomes are reportable is a narrow attentional bot

What carries the argument

The dynamic latent state, defined as the time-indexed weighting vector over the dimensions that process events into decisions and outcomes.

If this is right

  • Seven testable predictions follow directly from the state definition and its causal role.
  • State-aware systems must meet six operational requirements to perform effective interventions.
  • Applications in digital health become feasible by timing interventions to state trajectories.
  • AI personalisation shifts from static profiles to real-time state targeting.
  • Personal agency expands through deliberate state management at decision points.

Where Pith is reading between the lines

These are editorial extensions of the paper, not claims the author makes directly.

  • If the framework holds, self-tracking tools could focus on detecting and shifting the weighting vector rather than logging external events alone.
  • The sub-daily dynamism suggests pairing the approach with chronobiology data to schedule interventions by time of day.
  • In education settings the claim would imply designing feedback that adjusts for a learner's current state rather than fixed ability measures.
  • The attentional-bottleneck element points to interventions that first clear or redirect the narrow reportable channel before addressing downstream outcomes.

Load-bearing premise

The relationship between state, decision, and outcome is causal rather than merely correlational, and interventions can reliably reach the weighting vector at sub-daily timescales.

What would settle it

A controlled experiment that alters the weighting vector at decision time through an intervention yet produces no measurable shift in the predicted outcomes would falsify the controllability claim.

Figures

Figures reproduced from arXiv: 2605.27580 by Pritam Mukherjee, Saurav Gupta, Suraj Biswas.

Figure 1
Figure 1. Figure 1: Same observable input, different states, different outcomes. The event is held constant. The weighting vector S differs across three illustrative configurations. The outcome class diverges accordingly. The arrow from input to state is conditional rather than strictly causal because the state pre-exists the input. The arrow from state plus input to outcome is the causal claim [PITH_FULL_IMAGE:figures/full_… view at source ↗
Figure 2
Figure 2. Figure 2: depicts the causal path. State S_t propagates through three intermediate channels. Biology covers HPA￾axis dynamics, autonomic regulation, and endocrine signalling. Physiology covers heart-rate variability, sleep architecture, activity, and energy availability. Neuropsychology covers attention allocation, appraisal, and working memory. The three channels jointly shape the decision A_t, which produces an ou… view at source ↗
Figure 3
Figure 3. Figure 3: Within-day state oscillation across five illustrative individuals (schematic). The same composite state index varies through the day in patterns that share some structure (the early-morning trough) and diverge in others (afternoon dynamics). Between-person variation in the same dimension is large enough that group-level summaries discard most of the signal. 2.6 State has correlation structure that differs … view at source ↗
Figure 4
Figure 4. Figure 4: Illustrative state-dimension correlation structure across the four personas. The same dimensions are weighted differently in different groups, and the correlations among them carry information that a group average discards. The matrices are illustrative, not measurements. 3 Background and Related Work We organise the literature into six strands. We are deliberately brief on each, because the relevant 2020 … view at source ↗
Figure 5
Figure 5. Figure 5: The attentional bottleneck and state-dependent filtering. Sensory input is parallel and pre-attentive. Conscious processing is serial, capacity-limited, and biased. The state St controls what passes the bottleneck. Bandwidth estimates from Koch et al. [17], Dehaene [6], Lavie [19]. 4.2 What this implies for the verbal narrative A person who says, after a decision, "I closed the document because I was tired… view at source ↗
Figure 6
Figure 6. Figure 6: Observational base supporting the framework. (a) Cohort composition by persona across more than 200,000 consented participants observed over 24 months. (b) Cohort and study metadata. The deployment is observational and product-embedded. Aggregate qualitative findings are reported here. Quantitative analysis is deferred to a separate empirical companion [PITH_FULL_IMAGE:figures/full_fig_p009_6.png] view at source ↗
Figure 7
Figure 7. Figure 7: depicts, schematically, the kind of outcome-divergence pattern that is consistent with the framework. For a generic class of high-stakes feedback events, the distribution over outcome classes (withdrawal, rumination, reframing, action, integration) differs across the four personas. The pattern in [PITH_FULL_IMAGE:figures/full_fig_p010_7.png] view at source ↗
Figure 8
Figure 8. Figure 8: State trajectory across 24 hours, with intervention window. The trajectory is a schematic composite, not from production data. The point is structural. Not all moments are equally amenable to intervention. State-transition zones are windows in which a modest intervention can shift the trajectory. Outside those windows, the same intervention has small or no effect. The implication is that timing carries as … view at source ↗
Figure 9
Figure 9. Figure 9: Intervention timing and effect size: state-aware versus correlational delivery. The frontier is illustrative and qualitatively consistent with the just-in-time adaptive intervention literature [25]. The effect size at well-timed delivery is several times larger than at mistimed delivery. The correlational baseline cannot reach the peak because its trigger is engagement, not state. 5.7 State as a Markov str… view at source ↗
Figure 10
Figure 10. Figure 10: Simplified state-transition Markov structure (illustrative). Nodes are coarse state regions. Edge weights are illustrative transition probabilities. Red dashed rings mark transition zones in which intervention has high leverage. Real state dynamics are higher-dimensional and partly continuous, but the Markov picture captures the structural point: not all regions are equally controllable. 5.8 Three qualita… view at source ↗
Figure 11
Figure 11. Figure 11: What a state-aware system must operationally support. Six capability requirements (C1 to C6) follow from the state-causation framework. The right-most column suggests measurable signals by which a system can be audited against the requirements. C1. Real-time state estimation. The system must maintain an individual-indexed estimate of the current state at a cadence that matches the cadence at which states … view at source ↗
Figure 12
Figure 12. Figure 12: Forecast calibration over increasing per-individual observation depth. State-aware systems continue to improve as longitudinal data accumulates. Correlational baselines plateau and then degrade as inter-individual differences are absorbed into population estimates. The schematic frontier is the central testable signature of state￾aware design. C5. Counterfactual replay. The system must be able to reconstr… view at source ↗
read the original abstract

A central puzzle for the behavioural sciences and for human-facing artificial intelligence is the persistence of within-person variability. The same individual, presented with the same observable input, produces different outcomes on different occasions, and different individuals produce divergent outcomes that no observable covariate fully predicts. We argue that this variability belongs in the dynamic latent state of the person, and that human outcomes are controllable in a precise and operational sense through interventions that target the state and its weighting at the moment a decision is being formed. We define a state as the time-indexed weighting vector over the dimensions that govern how an individual's biology, physiology, and neuropsychology process the next event into a decision and an outcome. The relationship between state, decision, and outcome is causal rather than correlational. The weighting vector is dynamic at sub-daily timescales. The conscious channel through which outcomes are reportable is a narrow attentional bottleneck whose contents are themselves state-dependent. Taken together, these claims imply that the outcome of a given event is controllable, conditionally, on the state-trajectory at the time of intervention. We motivate the framework with six strands of established evidence (causal inference, predictive processing, allostasis, attentional bottleneck, chronobiology, computational psychiatry) and a 24-month observational base from a deployed behavioural platform spanning more than 200,000 consented users across four occupational personas (research period 2023 to 2026). We derive seven testable predictions, list six operational requirements for state-aware systems, and discuss implications for digital health, education, AI personalisation, and personal agency.

Editorial analysis

A structured set of objections, weighed in public.

Desk editor's note, referee report, simulated authors' rebuttal, and a circularity audit. Tearing a paper down is the easy half of reading it; the pith above is the substance, this is the friction.

Referee Report

3 major / 2 minor

Summary. The paper argues that within-person outcome variability arises from a dynamic latent state, defined as a time-indexed weighting vector over biological, physiological, and neuropsychological dimensions that processes events into decisions. It claims this state-outcome relationship is causal (not merely correlational), that the weighting vector varies at sub-daily timescales, and that targeted interventions on the state at decision formation can therefore control outcomes in a precise, operational sense. The framework is motivated by citations to six literatures (causal inference, predictive processing, allostasis, attentional bottleneck, chronobiology, computational psychiatry) plus a 24-month observational dataset from a deployed behavioral platform (>200k users across four personas); it derives seven testable predictions, lists six operational requirements for state-aware systems, and discusses implications for digital health, education, AI, and personal agency.

Significance. If the causal controllability claim can be secured with an explicit identification strategy, the framework would offer a unified, intervention-oriented account of human variability that integrates established literatures and could inform state-aware systems in AI personalization and digital health. The scale of the observational base (>200k users) is a potential strength for deriving falsifiable predictions, though the paper does not yet demonstrate that these predictions are independent of the model's own definitions.

major comments (3)
  1. [Abstract] Abstract: the foundational assertion that 'the relationship between state, decision, and outcome is causal rather than correlational' and that interventions 'target the state and its weighting at the moment a decision is being formed' is presented without an identification strategy (instrumental variables, regression discontinuity on state shifts, or within-person randomization) for the 24-month observational platform data. Observational covariation alone cannot secure the move to controllable causality given time-varying confounding and reverse causation risks.
  2. [Section on testable predictions] Section deriving the seven testable predictions: these predictions are generated directly from the state-as-weighting-vector definition and its sub-daily dynamics; without an independent empirical test (e.g., pre-registered intervention that alters the weighting vector and measures outcome change while holding observables fixed), they risk circularity and do not yet constitute load-bearing evidence for the controllability claim.
  3. [Framework definition] Framework definition of state (time-indexed weighting vector): the model treats the vector as both the latent cause and the target of intervention at sub-daily timescales, yet the manuscript provides no derivation or measurement protocol showing how the vector can be identified or manipulated independently of the outcome it is claimed to control.
minor comments (2)
  1. [Operational requirements] The six operational requirements for state-aware systems are listed but lack concrete implementation details (e.g., how the attentional bottleneck is operationalized in the platform data).
  2. [Framework] Notation for the dynamic weighting vector is introduced in the abstract but not carried through with explicit equations or variable definitions in the framework section, making the causal claims harder to evaluate formally.

Simulated Author's Rebuttal

3 responses · 0 unresolved

We thank the referee for these constructive comments, which help clarify the scope and evidentiary basis of our framework. We respond point-by-point below, indicating planned revisions where the manuscript can be strengthened without altering its core theoretical contribution.

read point-by-point responses
  1. Referee: [Abstract] Abstract: the foundational assertion that 'the relationship between state, decision, and outcome is causal rather than correlational' and that interventions 'target the state and its weighting at the moment a decision is being formed' is presented without an identification strategy (instrumental variables, regression discontinuity on state shifts, or within-person randomization) for the 24-month observational platform data. Observational covariation alone cannot secure the move to controllable causality given time-varying confounding and reverse causation risks.

    Authors: We agree the abstract states the causal claim directly. The causal interpretation is grounded in the integration of the six cited literatures rather than in a formal identification strategy applied to the observational dataset. The 24-month platform data is used to document scale and within-person variability and to motivate the seven predictions; it is not presented as an empirical demonstration of causality. We will revise the abstract and the opening of Section 2 to state explicitly that the framework advances a causal model whose empirical confirmation requires future designs with explicit identification strategies (e.g., within-person randomization or state-shift discontinuities). This revision will be made. revision: yes

  2. Referee: [Section on testable predictions] Section deriving the seven testable predictions: these predictions are generated directly from the state-as-weighting-vector definition and its sub-daily dynamics; without an independent empirical test (e.g., pre-registered intervention that alters the weighting vector and measures outcome change while holding observables fixed), they risk circularity and do not yet constitute load-bearing evidence for the controllability claim.

    Authors: The predictions are derived from the framework to render it falsifiable, yet we recognize that derivation alone does not constitute independent evidence. We will revise the section to (i) restate each prediction in terms of observable quantities that can be measured without presupposing the weighting vector, and (ii) outline concrete independent test designs (pre-registered within-person experiments and hold-out analyses on the platform data) that separate model construction from evaluation. This addresses the circularity concern while preserving the predictions as the framework's primary empirical output. revision: partial

  3. Referee: [Framework definition] Framework definition of state (time-indexed weighting vector): the model treats the vector as both the latent cause and the target of intervention at sub-daily timescales, yet the manuscript provides no derivation or measurement protocol showing how the vector can be identified or manipulated independently of the outcome it is claimed to control.

    Authors: The state is introduced as a latent theoretical construct synthesized from the cited literatures; the manuscript is a framework paper rather than an empirical measurement study. No identification or manipulation protocol is supplied because none has yet been developed for this specific vector. We will add a short subsection (new Section 3.4) that sketches candidate measurement routes drawn from computational psychiatry and chronobiology and that explicitly flags independent identification and manipulation as open empirical questions for follow-on work. This revision will be made. revision: yes

Circularity Check

1 steps flagged

Controllability asserted by definition of state as governing vector

specific steps
  1. self definitional [Abstract, paragraph 2]
    "We define a state as the time-indexed weighting vector over the dimensions that govern how an individual's biology, physiology, and neuropsychology process the next event into a decision and an outcome. The relationship between state, decision, and outcome is causal rather than correlational. The weighting vector is dynamic at sub-daily timescales. [...] Taken together, these claims imply that the outcome of a given event is controllable, conditionally, on the state-trajectory at the time of intervention."

    State is defined as the vector governing how inputs become decisions/outcomes; the causal relationship is asserted within the framework; controllability is then said to follow directly from these definitional claims. The result is equivalent to the input definition rather than independently derived.

full rationale

The paper defines state as the weighting vector that governs processing of events into decisions and outcomes, asserts the state-decision-outcome link is causal, and concludes controllability follows from these claims. This makes the central result true by the definitional setup rather than derived from independent evidence or identification. The seven predictions are presented as derived from the same framework. External literatures are cited for motivation, but the load-bearing move from definition to controllability reduces by construction. Partial circularity in the derivation chain.

Axiom & Free-Parameter Ledger

0 free parameters · 1 axioms · 1 invented entities

The central claim rests on the introduction of a new entity (the dynamic latent state) and the domain assumption of causality without independent evidence supplied in the abstract beyond the cited strands and platform data.

axioms (1)
  • domain assumption The relationship between state, decision, and outcome is causal rather than correlational
    Explicitly stated in the abstract as a foundational claim of the framework.
invented entities (1)
  • dynamic latent state defined as time-indexed weighting vector no independent evidence
    purpose: To account for within-person variability and serve as the target for causal interventions
    Newly defined in the abstract; no independent evidence or falsifiable handle provided beyond the framework itself.

pith-pipeline@v0.9.1-grok · 5822 in / 1389 out tokens · 56116 ms · 2026-06-29T17:06:46.371357+00:00 · methodology

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Forward citations

Cited by 1 Pith paper

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Reference graph

Works this paper leans on

42 extracted references · 1 canonical work pages · cited by 1 Pith paper

  1. [1]

    Abstract

    Biswas, Gupta, and Mukherjee · You Are in Control of Your State 1 / 20 You Are in Control of Your State: Why Human Outcomes Are Controllable Through Causal State Intervention Suraj Biswas · Saurav Gupta · Pritam Mukherjee ORCID: Suraj Biswas - https://orcid.org/0009-0008-1727-8179; Pritam Mukherjee - https://orcid.org/0009-0007-9018-4083 Independent Resea...

  2. [2]

    At the aggregate qualitative level, the observed patterns are consistent with the framework

    We describe a 24-month observational base from a deployed behavioural platform that spans more than 200,000 consented users across four occupational personas, with research carried out from 2023 to 2026 by the authors and their team. At the aggregate qualitative level, the observed patterns are consistent with the framework. We derive seven testable predi...

  3. [3]

    The same dimensions are weighted differently in different groups, and the correlations among them carry information that a group average discards

    Illustrative state-dimension correlation structure across the four personas. The same dimensions are weighted differently in different groups, and the correlations among them carry information that a group average discards. The matrices are illustrative, not measurements. 3 Background and Related Work We organise the literature into six strands. We are de...

  4. [4]

    The peer-reviewed numbers we cite are firmer

    and is widely repeated but not directly empirical. The peer-reviewed numbers we cite are firmer. The upper bound on conscious throughput is in the tens of bits per second in attention experiments [19, 20]. The neural-firing rate estimate is in Koch et al. [17]. The qualitative point survives. The gap is enormous, and what passes is state-dependent. 5 Obse...

  5. [5]

    We will report effect sizes, confidence intervals, and per-persona variance decomposition

    The disclosure boundary is the constraint. We will report effect sizes, confidence intervals, and per-persona variance decomposition. We will not report architectural details that fall within the scope of the patent applications. 9.2 Cross-deployment replication A single-deployment observational base cannot support the strongest claims the framework makes...

  6. [6]

    D., Ibeling, D., and Icard, T

    Bareinboim, E., Correa, J. D., Ibeling, D., and Icard, T. (2022). On Pearl’s hierarchy and the foundations of causal inference. In H. Geffner, R. Dechter, and J. Y. Halpern (Eds.), Probabilistic and Causal Inference: The Works of Judea Pearl (pp. 507–556). ACM Books

  7. [7]

    Biswas, S. (2026). Human modelling requires a causal architecture of behaviour and biology, not correlation. SSRN preprint

  8. [8]

    L., and Bjork, R

    Bjork, E. L., and Bjork, R. A. (2011). Making things hard on yourself, but in a good way: Creating desirable difficulties to enhance learning. In M. A. Gernsbacher et al. (Eds.), Psychology and the Real World (pp. 56–64). Worth Publishers

  9. [9]

    C., and Santamaría-García, H

    Castro Martínez, J. C., and Santamaría-García, H. (2023). Understanding mental health through computers: An introduction to computational psychiatry. Frontiers in Psychiatry, 14, 1092471

  10. [10]

    Cicchetti, D., and Rogosch, F. A. (1996). Equifinality and multifinality in developmental psychopathology. Development and Psychopathology, 8(4), 597–600

  11. [11]

    Dehaene, S. (2014). Consciousness and the brain: Deciphering how the brain codes our thoughts. Viking

  12. [12]

    Fleeson, W. (2001). Toward a structure- and process-integrated view of personality: Traits as density distributions of states. Journal of Personality and Social Psychology, 80(6), 1011–1027

  13. [13]

    Folkard, S., and Akerstedt, T. (2004). Trends in the risk of accidents and injuries and their implications for models of fatigue and performance. Aviation, Space, and Environmental Medicine, 75(3 Suppl), A161–A167

  14. [14]

    Friston, K. (2010). The free-energy principle: A unified brain theory? Nature Reviews Neuroscience, 11(2), 127–138

  15. [15]

    Friston, K. (2023). Computational psychiatry: From synapses to sentience. Molecular Psychiatry, 28(1), 256–268

  16. [16]

    S., and Kopin, I

    Goldstein, D. S., and Kopin, I. J. (2007). Evolution of concepts of stress. Stress, 10(2), 109–120

  17. [17]

    L., Asparouhov, T., Brose, A., Schmiedek, F., and Muthén, B

    Hamaker, E. L., Asparouhov, T., Brose, A., Schmiedek, F., and Muthén, B. (2018). At the frontiers of modeling intensive longitudinal data: Dynamic structural equation models for the affective measurements from the COGITO study. Multivariate Behavioral Research, 53(6), 820–841

  18. [18]

    Huys, Q. J. M., Maia, T. V., and Frank, M. J. (2016). Computational psychiatry as a bridge from neuroscience to clinical applications. Nature Neuroscience, 19(3), 404–413. Biswas, Gupta, and Mukherjee · You Are in Control of Your State 19 / 20

  19. [19]

    P., McEwen, B

    Juster, R. P., McEwen, B. S., and Lupien, S. J. (2010). Allostatic load biomarkers of chronic stress and impact on health and cognition. Neuroscience and Biobehavioral Reviews, 35(1), 2–16

  20. [20]

    Kahneman, D. (2011). Thinking, fast and slow. Farrar, Straus and Giroux

  21. [21]

    N., and McEwen, B

    Karatsoreos, I. N., and McEwen, B. S. (2011). Psychobiological allostasis: Resistance, resilience, and vulnerability. Trends in Cognitive Sciences, 15(12), 576–584

  22. [22]

    Koch, C., Massimini, M., Boly, M., and Tononi, G. (2016). Neural correlates of consciousness: Progress and problems. Nature Reviews Neuroscience, 17(5), 307–321

  23. [23]

    Kunda, Z. (1990). The case for motivated reasoning. Psychological Bulletin, 108(3), 480–498

  24. [24]

    Lavie, N. (1995). Perceptual load as a necessary condition for selective attention. Journal of Experimental Psychology: Human Perception and Performance, 21(3), 451–468

  25. [25]

    Lavie, N. (2005). Distracted and confused? Selective attention under load. Trends in Cognitive Sciences, 9(2), 75–82

  26. [26]

    B., Tzianas, L

    Limongi, R., Skelton, A. B., Tzianas, L. H., and Silva, A. M. (2024). Increasing the construct validity of computational phenotypes of mental illness through active inference and brain imaging. Brain Sciences, 14(12),

  27. [27]

    P., and Hasher, L

    May, C. P., and Hasher, L. (1998). Synchrony effects in inhibitory control over thought and action. Journal of Experimental Psychology: Human Perception and Performance, 24(2), 363–379

  28. [28]

    McEwen, B. S. (2007). Physiology and neurobiology of stress and adaptation: Central role of the brain. Physiological Reviews, 87(3), 873–904

  29. [29]

    Molenaar, P. C. M. (2004). A manifesto on psychology as idiographic science: Bringing the person back into scientific psychology, this time forever. Measurement: Interdisciplinary Research and Perspectives, 2(4), 201–218

  30. [30]

    N., Spring, B

    Nahum-Shani, I., Smith, S. N., Spring, B. J., Collins, L. M., Witkiewitz, K., Tewari, A., and Murphy, S. A. (2018). Just-in-time adaptive interventions (JITAIs) in mobile health. Annals of Behavioral Medicine, 52(6), 446–462

  31. [31]

    E., and Wilson, T

    Nisbett, R. E., and Wilson, T. D. (1977). Telling more than we can know: Verbal reports on mental processes. Psychological Review, 84(3), 231–259

  32. [32]

    Norretranders, T. (1998). The user illusion: Cutting consciousness down to size. Viking

  33. [33]

    Pearl, J. (2009). Causality: Models, reasoning, and inference (2nd ed.). Cambridge University Press

  34. [34]

    Pennycook, G., De Neys, W., Evans, J. S. B. T., Stanovich, K. E., and Thompson, V. A. (2018). The mythical dual-process typology. Trends in Cognitive Sciences, 22(8), 667–668

  35. [35]

    Pezzulo, G., Parr, T., Cisek, P., Clark, A., and Friston, K. (2024). Generating meaning: Active inference and the scope and limits of passive AI. Trends in Cognitive Sciences, 28(2), 97–112

  36. [36]

    Richens, J., and Everitt, T. (2024). Robust agents learn causal world models. ICLR 2024 (Outstanding Paper Honourable Mention). arXiv:2402.10877

  37. [37]

    T., Rivera, D

    Riley, W. T., Rivera, D. E., Atienza, A. A., Nilsen, W., Allison, S. M., and Mermelstein, R. (2011). Health behavior models in the age of mobile interventions: Are our theories up to the task? Translational Behavioral Medicine, 1(1), 53–71

  38. [38]

    L., and Karpicke, J

    Roediger, H. L., and Karpicke, J. D. (2006). Test-enhanced learning: Taking memory tests improves long-term retention. Psychological Science, 17(3), 249–255

  39. [39]

    Roenneberg, T., Wirz-Justice, A., and Merrow, M. (2003). Life between clocks: Daily temporal patterns of human chronotypes. Journal of Biological Rhythms, 18(1), 80–90

  40. [40]

    R., Kalchbrenner, N., Goyal, A., and Bengio, Y

    Schölkopf, B., Locatello, F., Bauer, S., Ke, N. R., Kalchbrenner, N., Goyal, A., and Bengio, Y. (2021). Toward causal representation learning. Proceedings of the IEEE, 109(5), 612–634

  41. [41]

    Sterling, P. (2012). Allostasis: A model of predictive regulation. Physiology and Behavior, 106(1), 5–15

  42. [42]

    D., and Dunn, E

    Wilson, T. D., and Dunn, E. W. (2004). Self-knowledge: Its limits, value, and potential for improvement. Annual Review of Psychology, 55, 493–518. Biswas, Gupta, and Mukherjee · You Are in Control of Your State 20 / 20 Author contributions S.B. conceived the framework, developed the formal definitions and the four-channel causal model, and led the writing...